EDTA
Ethylene
Diamine TetraAcetic
acid (EDTA) is a non-toxic amino acid that
was synthesized in Germany in 1931. It was
designed to treat severely lead poisoned
patients. Prior to its development, there
was little that could be done for these
unfortunate people. They died because of the
tremendous toxic effect of lead on the
brain, nervous system and other major
organs.
Chemists in the food
processing industry are quite familiar with
chelation chemistry and EDTA. The research
literature contains more than 3,000
reference papers concerning EDTA. EDTA is
used as a preservative in countless foods…
canned, bottled, and dry packed.
The chemistry of blood
banking is another source of EDTA
information. The substance is used in the
performance of many blood tests. Banked
blood has small amounts EDTA added to
prevent blood cells from breaking down. EDTA
is known to be a calcium blocking agent, a
potent coronary vasodilator. In other words,
EDTA can bind or chelate calcium as well as
other minerals in the body. It will remove
calcium particles deposited in arterial wall
plaques and atheromas. In addition, EDTA
blocks the slow calcium currents in the
arterial wall, resulting in arterial
vasodilation.
Probably the major underlying condition
leading to cardiovascular disease is
atherosclerosis, also known as hardening of
the arteries. This degenerative disease, in
time, can narrow or block arteries in the
heart, brain and other parts of the body. It
may begin early in life. The linings of the
arteries become thickened and roughened by
deposits of fat, cholesterol, fibrin (a
clotting material), cellular debris and
calcium.
As this buildup on the inner walls becomes
hard and thick, arteries lose their ability
to expand and contract. The blood moves with
difficulty through the narrowed artery
channels. This makes it easier for a clot to
form that will block the channel (lumen) and
deprive the heart, brain and other organs of
a necessary blood supply. In such a
situation, how can dilator drugs possibly be
effective?
When a complete blockage occurs in a vessel
to the brain, the result may be a cerebral
thrombosis, a form of stroke. Based on what
is known, scientists admit the relationship
between the amount of cholesterol and
saturated fats in the bloodstream, and
coronary artery disease a blockage of the
arteries that supply blood to the heart
muscle itself.
To review precisely what chelation is,
consider the following: The electromagnetic
attraction of fats and proteins for divalent
calcium that has wandered through the
injuries in blood vessel walls, is the same
process that enables EDTA to remove calcium
and fat from the plaque that occludes the
vessel. A study of over six hundred human
aortas has demonstrated alterations in the
elastic tissue with accumulations of calcium
prior to the deposition of fat and
cholesterol. (Blumenthal, 1944).
Calcium has two positive charges which are
called valences. Hence, calcium is divalent.
Calcium is strongly attracted
electromagnetically by the open ended,
molecular structure of EDTA that is
circulating in the blood during the
chelation treatment. This results in the
calcium ion being incorporated into the EDTA
molecular structure, forming a closed ring.
When this process takes place, the metal is
said to be chelated and EDTA is termed the
chelating agent.
When calcium (or other divalent metals such
as lead, mercury, cadmium, aluminum, etc.)
is chelated by EDTA, the original
electromagnetic attraction is lost, and the
fatty debris is dissolved by circulating
blood and metabolized. The calcium EDTA
molecule, now inactive and non toxic, is
carried by the blood until it passes through
the kidneys. It then is removed from the
body via the urine.
The solid sticky plaque goes into solution
and is harmlessly removed. By this unique
mechanism, dangerous solids are converted to
a liquid, then transported away to be
eliminated. This is a natural, normal
phenomenon of body chemistry.
Norman E. Clarke, Sr., M.D., a cardiologist
at Providence Hospital in Detroit, was the
first American to discover the many
beneficial effects of EDTA chelation. When
he treated battery factory workers for lead
poisoning, they reported relief of their
symptoms of chest pain (angina), arthritis,
intermittent claudication (severe leg pain
due to plugged arteries in the legs), as
well as their symptoms of lead poisoning.
Dr. Clarke, now in his eighties and very
active in practice and on the lecture
circuit, is recognized as a chelation
pioneer in the Soviet Union. The Russians
use chelation therapy as the second most
common treatment for arteriosclerotic artery
disease. It is also the preferred method of
treatment in Czechoslovakia. EDTA chelation
is administered with great success for blood
vessel disease, stroke, senility, diabetes,
kidney diseases, and other degenerative
diseases in Germany, Switzerland, Mexico and
Canada, to name just a few countries. -
Excerpts from "Chelation Can
Cure" by Dr. E.W. McDonagh
Summary Points
· EDTA
removes abnormally located metal ions, such
as copper and iron, that accumulate with
age.
·
EDTA removes lead, cadmium,
aluminum, and other metals, thereby
restoring enzyme systems to their proper
functions.
· EDTA
lowers blood calcium and thus stimulates the
production of parathormone from the
parathyroid glands. This mild pulse of
parathormone is responsible for the removal
of calcium from abnormal locations (such as
arteries) and the deposition of calcium in
locations (such as bones) where it should
be. This accounts for the mild
recalcification of osteoporotic bones seen
with EDTA.
· EDTA
stimulates the enlargement of small vessels,
so that they serve the purpose of collateral
circulation around a blockage, rendering the
blockage irrelevant.
· EDTA
controls free radical damage due to lipid
peroxidation by serving as a powerful
antioxidant.
· EDTA
enhances the integrity of cellular and
mitochondrial membranes.
· EDTA
helps reestablish prostaglandin hormone
balance.Prostaglandins, among other things,
are responsible for the balancing act
between contraction and relaxation of
arterial walls and between clotting and the
free flow of blood. Prostaglandins are
produced from fatty acids, therefore lipid
peroxidation upsets the balance of these
vital hormones. EDTA chelates out the
catalyzing metallic co-enzymes and thus
inhibits lipid peroxidation, also serving
the same function as an antioxidant.
· EDTA
reduces the tendency of platelets to cause
coagulation too readily. This tends to
prevent inappropriate thrombosis, which
blocks coronary arteries during a heart
attack.
·
EDTA increases tissue
flexibility by uncoupling age-related
cross-linkages that are responsible for loss
of skin tone and for wrinkling